tag:blogger.com,1999:blog-4082643038671225434.post8786630464789495726..comments2023-10-07T01:34:01.774-07:00Comments on Case of the Day: Nerve IICraighttp://www.blogger.com/profile/17072102331564743101noreply@blogger.comBlogger1125tag:blogger.com,1999:blog-4082643038671225434.post-42963373241487938132010-09-13T20:29:36.138-07:002010-09-13T20:29:36.138-07:00Nerve II
This is refeeding syndrome, resulting fr...Nerve II<br /><br />This is refeeding syndrome, resulting from rapid fluid and electrolyte changes usually within the first 2 weeks of refeeding when patients who are malnourished are given oral, enteral, or parenteral feedings. Malnourished patients have depleted intracellular phosphate stores. With refeeding, metabolism shifts from fat to carbohydrates which can induce hypophosphatemia (as a result of glycolysis). Hypophosphatemia leads to impaired energy stores due to depletion of intracellular ATP and tissue hypoxia due to reduced levels of 2,3-DPG. In addition, hypomagnesemia and hypokalemia can lead to arrythmias and Wernicke's encephalopathy can occur in thiamine-deficient patients.<br /><br />Source: UpToDate.Craighttps://www.blogger.com/profile/17072102331564743101noreply@blogger.com